Hemostaza pierwotna pdf free. Information of the substance preparation and of the company. The most comprehensive portfolio of hemostasis solutions. HEMOSTAZA . DZIECI: profilaktyka pierwotna; obecnie do 18 r.ż. starania PSCH o przedłużenie do 26 r.ź. leki osoczopochodne /.

Author: Mir Meztilrajas
Country: Venezuela
Language: English (Spanish)
Genre: Finance
Published (Last): 2 December 2006
Pages: 444
PDF File Size: 11.41 Mb
ePub File Size: 17.47 Mb
ISBN: 301-8-19298-861-8
Downloads: 1695
Price: Free* [*Free Regsitration Required]
Uploader: Samuk

And both of these are provided by the platelets. Charakterystyka epidemiologicznokliniczna boreliozy z lyme w regionie polnocnowschodnim polski. It’s underneath the endothelial cells. To assess the presence of free fluid blood in the body cavities one can use mobile ultrasound fast focused assessment with sonography for trauma.

So what will happen is anti-thrombin III will interact with this heparin-like molecule. The first stage is primary hemostasis.

This protein is not normally exposed to the blood. And this fibrin mesh over the platelet plug is what we call a clot. They both do the same thing. When we have thrombin and thrombomodulin working together and interacting with each other, then now protein C, with the pierwotba of protein S will get activated. And now in anticoagulation, we have anti-thrombin III, and we want to prevent a clot.

So our bodies need a way to make sure pierwotba don’t get many clots. Naleza do nich pierwotna pierwszoerzedowa okolica ruchowa, okolica przedruchowa i dodatkowa okolica ruchowa.

So here we have our platelets that are floating around in our blood.


Antykoagulacja i tromboliza (film) | Khan Academy

Pozar microwave engineering e books electrical engineering. Which, is about 93 thousand miles.

What happens is, the endothelial cells secrete these proteins and send a signal to the platelets to come to that sight of injury. But fibrin isn’t floating around in our blood as fibrin. So you can see, in hemostasis we have thrombin and we want to make a clot. And when it has this extra piece of protein, it’s called fibrinogen. But plasmin isn’t floating around in our blood all the time, or else we wouldn’t be able to clot at all, because plasmin would be breaking down fibrin and fibrinogen all the time.

I like to think of it as, like a little shark floating around waiting to break down those clots. I’m drawing them all in one color, but there are several different types of coagulation factors. So here we have our clotting factors, the family of proteins. And so we get plasmin from plasminogen. So, we need a way of getting fibrin from fibrinogen.

So, let me erase this. And what this molecule does is, it interacts with another protein that’s already floating around in our blood. One of the most important ones is our red blood cell that carries oxygen all over to all our tissues in our body. So now that we have fibrin. Hemostaza uwalniana z ziarnistosci alfa plytek krwi trombina, powoduje wytworzenie fibryny z fibrynogenu. So let’s talk about anticoagulation first. And that’s important because we want to make sure that our blood vessels stay open and blood is able to flow through smoothly.


This sends signals to the blood that there’s been an injury.

Hemostaza wtórna

So we want to make sure we prevent clots from happening. And this is important because it links making the fibrin mesh with the platelet plug. The reason I drew squares is I want to make a note of what they carry inside of them. And we need plasmin whenever we don’t want to make a clot or whenever we don’t need to make pierwotnz clot. So, let me erase everything. So, here we have a blood vessel, and in our blood vessel we have our blood.

And the other thing that they do is they act on the smooth muscle cells of the blood vessel, and they cause vasodilation.

Hemostaza pierwotna pdf free

So, you can see our blood vessels have a lot of work to do. And ultimately what we end up with is we get fibrin from fibrinogen.

We do that through a process called hemostasis.